I received my PhD in Molecular and Cellular Biology at the University of Massachusetts Amherst in September 2012. During my graduate studies, I became interested in phenotypic and clinical difference between estrogen receptor alpha positive and negative breast cancers. Over time, my research interests broadened to how dysregulation of different proteins and pathways results in development and progression of aggressive breast cancer subtypes.

I came to North Carolina to work in the Fleming Lab because it was a great opportunity to continue my training as a breast cancer biologist and to fine tune the key characteristics that make a good scientist: integrity, passion, curiosity, and a quest for knowledge.

My career goals involve contributing the eradication of cancer by becoming a research scientists studying the biochemical pathways and molecular systems critical in the etiology of cancer in collaboration with oncologists and pathologists. I am committed to my goals for my future of running my own lab to study the molecular mechanisms involved in carcinogenesis

I received my PhD in Molecular and Cellular Biology at the University of Massachusetts Amherst in September 2012. During my graduate studies, I became interested in phenotypic and clinical difference between estrogen receptor alpha positive and negative breast cancers. Over time, my research interests broadened to how dysregulation of different proteins and pathways results in development and progression of aggressive breast cancer subtypes. I came to North Carolina to work in the Fleming Lab because it was a great opportunity to continue my training as a breast cancer biologist and to fine tune the key characteristics that make a good scientist: integrity, passion, curiosity, and a quest for knowledge. My career goals involve contributing the eradication of cancer by becoming a research scientists studying the biochemical pathways and molecular systems critical in the etiology of cancer in collaboration with oncologists and pathologists. I am committed to my goals for my future of running my own lab to study the molecular mechanisms involved in carcinogenesis

PhD	University of Massachusetts, Amherst	2013
BS	State University of New York, Albany	2004

1.	Fagan-Solis, K. D., & Smith Schneider , S. , & Pentecost, B. T., & Bentley, B. A. (2012). The ras homolog gene family, member A (RhoA) pathway mediates MMP-2 and MMP-9-independent invasive behavior in a triple-negative breast cancer cell line. Journal of Cellular Biochemistry.
2.	Turk, C. M., & Fagan-Solis, K. D., & Paczkowski, K. E., & Gozgit, J. M. (2012). Paralemmin-1 is over-expressed in estrogen-receptor positive breast cancers. Cancer Cell International, 12, 17.
3.	Moffatt, L. T., & Fagan-Solis, K. D., & Browne, E. P., & Arcaro, K. F. (2011). Vitellogenin and 11? hydroxylase mRNA expression in male Japanese medaka (Oryzias latipes) exposed in a short term laboratory assay to low levels of three estrogenic contaminants. International Society of Environmental Indicators, 1.